Modeling of Cisplatin-Induced Signaling Dynamics in Triple-Negative Breast Cancer Cells Reveals Mediators of Sensitivity-Reference-Cited by-同舟云学术

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Modeling of Cisplatin-Induced Signaling Dynamics in Triple-Negative Breast Cancer Cells Reveals Mediators of Sensitivity

Published:2019-08 Issue:9 Volume:28 Page:2345-2357.e5
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Heijink Anne Margriet,Everts Marieke,Honeywell Megan E.,Richards Ryan,Kok Yannick P.,de Vries Elisabeth G.E.,Lee Michael J.,van Vugt Marcel A.T.M.

Funder

Netherlands Organization for Scientific Research

European Research Council

Dutch Cancer Society

René Vogels Foundation

De Boer-Merema Foundation

American Cancer Society

NIGMS/NIH

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference46 articles.

1. The breast cancer susceptibility gene BRCA1 is required for subnuclear assembly of Rad51 and survival following treatment with the DNA cross-linking agent cisplatin;Bhattacharyya;J. Biol. Chem.,2000

2. Cisplatin sensitivity is related to late DNA damage processing and checkpoint control rather than to the early DNA damage response;Brozovic;Mutat. Res.,2009

3. Pathologic complete response rates in young women with BRCA1-positive breast cancers after neoadjuvant chemotherapy;Byrski;J. Clin. Oncol.,2010

4. 3rd ESO-ESMO International Consensus Guidelines for Advanced Breast Cancer (ABC 3);Cardoso;Ann. Oncol.,2017

5. The DNA damage response: making it safe to play with knives;Ciccia;Mol. Cell,2010

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