Trafficking-Mediated STING Degradation Requires Sorting to Acidified Endolysosomes and Can Be Targeted to Enhance Anti-tumor Response
Author:
Funder
NIH
Burroughs Wellcome Fund
Publisher
Elsevier BV
Subject
General Biochemistry, Genetics and Molecular Biology
Reference21 articles.
1. Cytosolic-DNA-mediated, STING-dependent proinflammatory gene induction necessitates canonical NF-κB activation through TBK1;Abe;J. Virol.,2014
2. Cutting edge: antimalarial drugs inhibit IFN-β production through blockade of cyclic GMP-AMP synthase-DNA interaction;An;J. Immunol.,2015
3. Direct activation of STING in the tumor microenvironment leads to potent and systemic tumor regression and immunity;Corrales;Cell Rep.,2015
4. STING activation by translocation from the ER is associated with infection and autoinflammatory disease;Dobbs;Cell Host Microbe,2015
5. Effects of small molecules on chaperone-mediated autophagy;Finn;Autophagy,2005
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