PRMT5 Is a Critical Regulator of Breast Cancer Stem Cell Function via Histone Methylation and FOXP1 Expression-Reference-Cited by-同舟云学术

PRMT5 Is a Critical Regulator of Breast Cancer Stem Cell Function via Histone Methylation and FOXP1 Expression

Published:2017-12 Issue:12 Volume:21 Page:3498-3513
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Chiang Kelly,Zielinska Agnieszka E.,Shaaban Abeer M.,Sanchez-Bailon Maria Pilar,Jarrold James,Clarke Thomas L.,Zhang Jingxian,Francis Adele,Jones Louise J.,Smith Sally,Barbash Olena,Guccione Ernesto,Farnie Gillian,Smalley Matthew J.,Davies Clare C.

Funder

University of Birmingham

Breast Cancer Now

Medical Research Council

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

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3. PRMT5-PTEN molecular pathway regulates senescence and self-renewal of primary glioblastoma neurosphere cells;Banasavadi-Siddegowda;Oncogene,2017

4. Regulation of constitutive and alternative splicing by PRMT5 reveals a role for Mdm4 pre-mRNA in sensing defects in the spliceosomal machinery;Bezzi;Genes Dev.,2013

5. Arginine methylation by PRMT1 regulates muscle stem cell fate;Blanc;Mol. Cell. Biol.,2017

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