Poly(ADP-ribosyl)ation of TIMELESS limits DNA replication stress and promotes stalled fork protection
Author:
Funder
National Institutes of Health
American Cancer Society
Publisher
Elsevier BV
Reference63 articles.
1. The multifaceted roles of PARP1 in DNA repair and chromatin remodelling;Ray Chaudhuri;Nat. Rev. Mol. Cell Biol.,2017
2. PARP-1 Activation Requires Local Unfolding of an Autoinhibitory Domain;Dawicki-McKenna;Mol. Cell,2015
3. Structural Basis of Detection and Signaling of DNA Single-Strand Breaks by Human PARP-1;Eustermann;Mol. Cell,2015
4. Importance of poly(ADP-ribose) glycohydrolase in the control of poly(ADP-ribose) metabolism;Davidovic;Exp. Cell Res.,2001
5. HPF1 completes the PARP active site for DNA damage-induced ADP-ribosylation;Suskiewicz;Nature,2020
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1. USP37 protects mammalian cells during DNA replication stress by counteracting CUL2LRR1and TRAIP;2024-09-03
2. PARP1-driven repair of topoisomerase IIIα DNA-protein crosslinks by FEN1;Cell Reports;2024-08
3. Synthetic lethality by PARP inhibitors: new mechanism uncovered based on unresolved transcription-replication conflicts;Signal Transduction and Targeted Therapy;2024-07-29
4. Intrinsic PARG inhibitor sensitivity is mimicked by TIMELESS haploinsufficiency and rescued by nucleoside supplementation;NAR Cancer;2024-07-09
5. The TIMELESS and PARP1 interaction suppresses replication-associated DNA gap accumulation;Nucleic Acids Research;2024-05-27
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