Flavivirus NS1 Triggers Tissue-Specific Vascular Endothelial Dysfunction Reflecting Disease Tropism-Reference-Cited by-同舟云学术

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Flavivirus NS1 Triggers Tissue-Specific Vascular Endothelial Dysfunction Reflecting Disease Tropism

Published:2019-02 Issue:6 Volume:26 Page:1598-1613.e8
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Puerta-Guardo Henry,Glasner Dustin R.,Espinosa Diego A.,Biering Scott B.,Patana Mark,Ratnasiri Kalani,Wang Chunling,Beatty P. Robert,Harris Eva

Funder

NIH

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference50 articles.

1. Cathepsin L is responsible for processing and activation of proheparanase through multiple cleavages of a linker segment;Abboud-Jarrous;J. Biol. Chem.,2008

2. The secreted form of dengue virus nonstructural protein NS1 is endocytosed by hepatocytes and accumulates in late endosomes: implications for viral infectivity;Alcon-LePoder;J. Virol.,2005

3. Vascular leakage in severe dengue virus infections: a potential role for the nonstructural viral protein NS1 and complement;Avirutnan;J. Infect. Dis.,2006

4. Antagonism of the complement component C4 by flavivirus nonstructural protein NS1;Avirutnan;J. Exp. Med.,2010

5. Tropism of dengue virus in mice and humans defined by viral nonstructural protein 3-specific immunostaining;Balsitis;Am. J. Trop. Med. Hyg.,2009

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