IL-23 and IL-1β Drive Human Th17 Cell Differentiation and Metabolic Reprogramming in Absence of CD28 Costimulation-Reference-Cited by-同舟云学术

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IL-23 and IL-1β Drive Human Th17 Cell Differentiation and Metabolic Reprogramming in Absence of CD28 Costimulation

Published:2018-03 Issue:10 Volume:22 Page:2642-2653
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Revu Shankar,Wu Jing,Henkel Matthew,Rittenhouse Natalie,Menk Ashley,Delgoffe Greg M.,Poholek Amanda C.,McGeachy Mandy J.

Funder

Rheumatology Research Foundation

Stand Up 2 Cancer-American Association of Cancer Research

NIH

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference77 articles.

1. IL-17 signaling: the yin and the yang;Amatya;Trends Immunol.,2017

2. The phenotype of human Th17 cells and their precursors, the cytokines that mediate their differentiation and the role of Th17 cells in inflammation;Annunziato;Int. Immunol.,2008

3. Human and murine Th17;Annunziato;Curr. Opin. HIV AIDS,2010

4. Metabolic control of the Treg/Th17 axis;Barbi;Immunol. Rev.,2013

5. The costimulatory molecule ICOS regulates the expression of c-Maf and IL-21 in the development of follicular T helper cells and TH-17 cells;Bauquet;Nat. Immunol.,2009

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