Age-Associated Impairments in Mitochondrial ADP Sensitivity Contribute to Redox Stress in Senescent Human Skeletal Muscle-Reference-Cited by-同舟云学术

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Age-Associated Impairments in Mitochondrial ADP Sensitivity Contribute to Redox Stress in Senescent Human Skeletal Muscle

Published:2018-03 Issue:11 Volume:22 Page:2837-2848
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Holloway Graham P.,Holwerda Andrew M.,Miotto Paula M.,Dirks Marlou L.,Verdijk Lex B.,van Loon Luc J.C.

Funder

Natural Sciences and Engineering Research Council of Canada

TI Food and Nutrition

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference68 articles.

1. Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo;Amara;Proc. Natl. Acad. Sci. U S A,2007

2. Type II skeletal myofibers possess unique properties that potentiate mitochondrial H(2)O(2) generation;Anderson;Am. J. Physiol. Cell Physiol.,2006

3. Induction of endogenous uncoupling protein 3 suppresses mitochondrial oxidant emission during fatty acid-supported respiration;Anderson;J. Biol. Chem.,2007

4. Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans;Anderson;J. Clin. Invest.,2009

5. Qualitative and quantitative changes in skeletal muscle mtDNA and expression of mitochondrial-encoded genes in the human aging process;Barrientos;Biochem. Mol. Med.,1997

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