TLR Crosstalk Activates LRP1 to Recruit Rab8a and PI3Kγ for Suppression of Inflammatory Responses-Reference-Cited by-同舟云学术

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TLR Crosstalk Activates LRP1 to Recruit Rab8a and PI3Kγ for Suppression of Inflammatory Responses

Published:2018-09 Issue:11 Volume:24 Page:3033-3044
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Luo Lin,Wall Adam A.,Tong Samuel J.,Hung Yu,Xiao Zhijian,Tarique Abdullah A.,Sly Peter D.,Fantino Emmanuelle,Marzolo María-Paz,Stow Jennifer L.

Funder

National Health and Medical Research Council of Australia

FONDECYT of Chile

Discovery Early Career Researcher Award from the Australian Research Council

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference50 articles.

1. Lipopolysaccharide interaction with cell surface Toll-like receptor 4-MD-2: higher affinity than that with MD-2 or CD14;Akashi;J. Exp. Med.,2003

2. The p110δ isoform of the kinase PI(3)K controls the subcellular compartmentalization of TLR4 signaling and protects from endotoxic shock;Aksoy;Nat. Immunol.,2012

3. The NPXY internalization signal of the LDL receptor adopts a reverse-turn conformation;Bansal;Cell,1991

4. miRNA-155 controls mast cell activation by regulating the PI3Kγ pathway and anaphylaxis in a mouse model;Biethahn;Allergy,2014

5. Signaling through LRP1: protection from atherosclerosis and beyond;Boucher;Biochem. Pharmacol.,2011

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