A Splicing-Independent Function of RBM10 Controls Specific 3′ UTR Processing to Regulate Cardiac Hypertrophy-Reference-Cited by-同舟云学术

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A Splicing-Independent Function of RBM10 Controls Specific 3′ UTR Processing to Regulate Cardiac Hypertrophy

Published:2018-09 Issue:13 Volume:24 Page:3539-3553
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Mohan Nimmy,Kumar Vikas,Kandala Divya T.,Kartha Chandrasekharan C.,Laishram Rakesh S.

Funder

Wellcome Trust-DBT India Alliance

Science and Engineering Research Board

Ministry of Science and Technology

Council of Scientific and Industrial Research

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference45 articles.

1. Rbm10 regulates inflammation development via alternative splicing of Dnmt3b;Atsumi;Int. Immunol.,2017

2. [Cardiac hypertrophy: molecular and cellular events];Carreño;Rev. Esp. Cardiol.,2006

3. Genome-Wide Polyadenylation Maps Reveal Dynamic mRNA 3′-End Formation in the Failing Human Heart;Creemers;Circ. Res.,2016

4. Phenotyping hypertrophy: eschew obfuscation;Dorn;Circ. Res.,2003

5. Cardiac hypertrophy: the good, the bad, and the ugly;Frey;Annu. Rev. Physiol.,2003

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