Modeling Late-Onset Sporadic Alzheimer’s Disease through BMI1 Deficiency-Reference-Cited by-同舟云学术

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Modeling Late-Onset Sporadic Alzheimer’s Disease through BMI1 Deficiency

Published:2018-05 Issue:9 Volume:23 Page:2653-2666
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Flamier Anthony,El Hajjar Jida,Adjaye James,Fernandes Karl J.,Abdouh Mohamed,Bernier Gilbert

Funder

Canadian Institutes of Health Research

National Science and Engineering Research Council of Canada

Molecular Biology Program of Université de Montréal

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference50 articles.

1. BMI1 sustains human glioblastoma multiforme stem cell renewal;Abdouh;J. Neurosci.,2009

2. Alzheimer’s disease hyperphosphorylated tau sequesters normal tau into tangles of filaments and disassembles microtubules;Alonso;Nat. Med.,1996

3. Regulation of HSF1 function in the heat stress response: implications in aging and disease;Anckar;Annu. Rev. Biochem.,2011

4. N-cadherin regulates p38 MAPK signaling via association with JNK-associated leucine zipper protein: implications for neurodegeneration in Alzheimer disease;Ando;J. Biol. Chem.,2011

5. Brain choline acetyltransferase and mental function in Alzheimer disease;Baskin;Arch. Neurol.,1999

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