Th1 Differentiation Drives the Accumulation of Intravascular, Non-protective CD4 T Cells during Tuberculosis-Reference-Cited by-同舟云学术

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Th1 Differentiation Drives the Accumulation of Intravascular, Non-protective CD4 T Cells during Tuberculosis

Published:2017-03 Issue:13 Volume:18 Page:3091-3104
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Sallin Michelle A.,Sakai Shunsuke,Kauffman Keith D.,Young Howard A.,Zhu Jinfang,Barber Daniel L.

Funder

Intramural Research Program

NIAID/NIH

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference33 articles.

1. Continuous effector CD8(+) T cell production in a controlled persistent infection is sustained by a proliferative intermediate population;Chu;Immunity,2016

2. Interleukin 12 (IL-12) is crucial to the development of protective immunity in mice intravenously infected with mycobacterium tuberculosis;Cooper;J. Exp. Med.,1997

3. Pathological role of interleukin 17 in mice subjected to repeated BCG vaccination after infection with Mycobacterium tuberculosis;Cruz;J. Exp. Med.,2010

4. Interferon gamma eliminates responding CD4 T cells during mycobacterial infection by inducing apoptosis of activated CD4 T cells;Dalton;J. Exp. Med.,2000

5. IL-12 increases resistance of BALB/c mice to Mycobacterium tuberculosis infection;Flynn;J. Immunol.,1995

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