Excess Synaptojanin 1 Contributes to Place Cell Dysfunction and Memory Deficits in the Aging Hippocampus in Three Types of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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Excess Synaptojanin 1 Contributes to Place Cell Dysfunction and Memory Deficits in the Aging Hippocampus in Three Types of Alzheimer’s Disease

Published:2018-06 Issue:10 Volume:23 Page:2967-2975
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Miranda Andre M.,Herman Mathieu,Cheng Rong,Nahmani Eden,Barrett Geoffrey,Micevska Elizabeta,Fontaine Gaelle,Potier Marie-Claude,Head Elizabeth,Schmitt Frederick A.,Lott Ira T.,Jiménez-Velázquez Ivonne Z.,Antonarakis Stylianos E.,Di Paolo Gilbert,Lee Joseph H.,Hussaini S. Abid,Marquer Catherine

Funder

Fundação para a Ciência e Tecnologia

Philippe Chatrier Foundation

Lejeune Foundation

Alzheimer’s Association

ANR Investissements d’Avenir

NIH

BrightFocus Foundation

NIA

NIH/NIA

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference59 articles.

1. A homozygous potentially pathogenic variant in the PAXBP1 gene in a large family with global developmental delay and myopathic hypotonia;Alharby;Clin. Genet.,2017

2. Down syndrome and the complexity of genome dosage imbalance;Antonarakis;Nat. Rev. Genet.,2017

3. Excessive expression of synaptojanin in brains with Down syndrome;Arai;Brain Dev.,2002

4. A founder mutation in presenilin 1 causing early-onset Alzheimer disease in unrelated Caribbean Hispanic families;Athan;JAMA,2001

5. Zebrafish ciliopathy screen plus human mutational analysis identifies C21orf59 and CCDC65 defects as causing primary ciliary dyskinesia;Austin-Tse;Am. J. Hum. Genet.,2013

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