Constitutive Interferon Maintains GBP Expression Required for Release of Bacterial Components Upstream of Pyroptosis and Anti-DNA Responses-Reference-Cited by-同舟云学术

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Constitutive Interferon Maintains GBP Expression Required for Release of Bacterial Components Upstream of Pyroptosis and Anti-DNA Responses

Published:2018-07 Issue:1 Volume:24 Page:155-168.e5
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Liu Beiyun C.,Sarhan Joseph,Panda Alexander,Muendlein Hayley I.,Ilyukha Vladimir,Coers Jörn,Yamamoto Masahiro,Isberg Ralph R.,Poltorak Alexander

Funder

NIH

Russian Science Fund Project

HHMI

NIAID

Government Contract

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference72 articles.

1. Caspase-11 protects against bacteria that escape the vacuole;Aachoui;Science,2013

2. Commensal bacteria calibrate the activation threshold of innate antiviral immunity;Abt;Immunity,2012

3. STING manifests self DNA-dependent inflammatory disease;Ahn;Proc. Natl. Acad. Sci. USA,2012

4. The frustrated host response to Legionella pneumophila is bypassed by MyD88-dependent translation of pro-inflammatory cytokines;Asrat;PLoS Pathog.,2014

5. IL-1α signaling initiates the inflammatory response to virulent Legionella pneumophila in vivo;Barry;J. Immunol.,2013

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1. Iron-depleting nutritional immunity controls extracellular bacterial replication in Legionella pneumophila infections;Nature Communications;2024-09-08

2. TrxR1 is involved in the activation of Caspase-11 by regulating the oxidative-reductive status of Trx-1;Redox Biology;2024-09

3. Mouse guanylate-binding proteins of the chromosome 3 cluster do not mediate antiviral activity in vitro or in mouse models of infection;Communications Biology;2024-08-25

4. Innate immune responses and monocyte-derived phagocyte recruitment in protective immunity to pathogenic bacteria: insights from Legionella pneumophila;Current Opinion in Microbiology;2024-08

5. Caspase-11 drives macrophage hyperinflammation in models of Polg-related mitochondrial disease;2024-05-14

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