Chronic Optogenetic Activation Augments Aβ Pathology in a Mouse Model of Alzheimer Disease-Reference-Cited by-同舟云学术

Chronic Optogenetic Activation Augments Aβ Pathology in a Mouse Model of Alzheimer Disease

Published:2015-05 Issue:6 Volume:11 Page:859-865
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Yamamoto Kaoru,Tanei Zen-ichi,Hashimoto Tadafumi,Wakabayashi Tomoko,Okuno Hiroyuki,Naka Yasushi,Yizhar Ofer,Fenno Lief E.,Fukayama Masashi,Bito Haruhiko,Cirrito John R.,Holtzman David M.,Deisseroth Karl,Iwatsubo Takeshi

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference18 articles.

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3. Synaptic activity regulates interstitial fluid amyloid-β levels in vivo;Cirrito;Neuron,2005

4. Endocytosis is required for synaptic activity-dependent release of amyloid-β in vivo;Cirrito;Neuron,2008

5. Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein;Games;Nature,1995

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