The Presenilin-1 ΔE9 Mutation Results in Reduced γ-Secretase Activity, but Not Total Loss of PS1 Function, in Isogenic Human Stem Cells-Reference-Cited by-同舟云学术

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The Presenilin-1 ΔE9 Mutation Results in Reduced γ-Secretase Activity, but Not Total Loss of PS1 Function, in Isogenic Human Stem Cells

Published:2013-11 Issue:4 Volume:5 Page:974-985
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Woodruff Grace,Young Jessica E.,Martinez Fernando J.,Buen Floyd,Gore Athurva,Kinaga Jennifer,Li Zhe,Yuan Shauna H.,Zhang Kun,Goldstein Lawrence S.B.

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference51 articles.

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2. The projected effect of risk factor reduction on Alzheimer’s disease prevalence;Barnes;Lancet Neurol.,2011

3. Presenilin clinical mutations can affect gamma-secretase activity by different mechanisms;Bentahir;J. Neurochem.,2006

4. CSF phosphorylated tau protein correlates with neocortical neurofibrillary pathology in Alzheimer’s disease;Buerger;Brain,2006

5. Highly efficient neural conversion of human ES and iPS cells by dual inhibition of SMAD signaling;Chambers;Nat. Biotechnol.,2009

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