MLKL and FADD Are Critical for Suppressing Progressive Lymphoproliferative Disease and Activating the NLRP3 Inflammasome-Reference-Cited by-同舟云学术

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MLKL and FADD Are Critical for Suppressing Progressive Lymphoproliferative Disease and Activating the NLRP3 Inflammasome

Published:2016-09 Issue:12 Volume:16 Page:3247-3259
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Zhang Xixi,Fan Cunxian,Zhang Haiwei,Zhao Qun,Liu Yongbo,Xu Chengxian,Xie Qun,Wu Xiaoxia,Yu Xianjun,Zhang Jianke,Zhang Haibing

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference52 articles.

1. NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder;Agostini;Immunity,2004

2. Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression;Bauernfeind;J. Immunol.,2009

3. Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death;Boldin;Cell,1996

4. The adaptor protein FADD protects epidermal keratinocytes from necroptosis in vivo and prevents skin inflammation;Bonnet;Immunity,2011

5. Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis;Cai;Nat. Cell Biol.,2014

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