Ronin Governs Early Heart Development by Controlling Core Gene Expression Programs-Reference-Cited by-同舟云学术

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Ronin Governs Early Heart Development by Controlling Core Gene Expression Programs

Published:2017-11 Issue:6 Volume:21 Page:1562-1573
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Fujita Jun,Freire Pablo,Coarfa Cristian,Benham Ashley L.,Gunaratne Preethi,Schneider Michael D.,Dejosez Marion,Zwaka Thomas P.

Funder

Cancer Prevention and Research Institute of Texas

Huffington Foundation

NIH

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference44 articles.

1. A new role of Ronin (Thap11) in the neural crest and craniofacial development in the mouse;Achilleos;FASEB J.,2017

2. Ronin (Thap11) is implicated in a new cobalamin deficiency syndrome impacting the central nervous system;Achilleos;FASEB J.,2017

3. Gene recombination in postmitotic cells. Targeted expression of Cre recombinase provokes cardiac-restricted, site-specific rearrangement in adult ventricular muscle in vivo;Agah;J. Clin. Invest.,1997

4. THAP5 is a human cardiac-specific inhibitor of cell cycle that is cleaved by the proapoptotic Omi/HtrA2 protease during cell death;Balakrishnan;Am. J. Physiol. Heart Circ. Physiol.,2009

5. Histone deacetylases 5 and 9 govern responsiveness of the heart to a subset of stress signals and play redundant roles in heart development;Chang;Mol. Cell. Biol.,2004

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1. Generation of human pluripotent stem cell lines (WAe009-A) with THAP11F80L cobalamin disorder-associated mutation;Stem Cell Research;2024-09

2. Lpt, trr, and Hcf regulate histone mono- and dimethylation that are essential for Drosophila heart development;Developmental Biology;2022-10

3. Glucosylceramide synthase inhibition protects against cardiac hypertrophy in chronic kidney disease;Scientific Reports;2022-06-04

4. MYC integrates FSH signalling networks in cumulus cells during bovine oocyte maturation;Acta Veterinaria Hungarica;2022-05-02

5. Mutations in Hcfc1 and Ronin result in an inborn error of cobalamin metabolism and ribosomopathy;Nature Communications;2022-01-10

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