FANCD2-dependent mitotic DNA synthesis relies on PCNA K164 ubiquitination
Author:
Funder
National Institutes of Health
National Cancer Institute
National Institute of General Medical Sciences
Achievement Rewards for College Scientists Foundation
Publisher
Elsevier BV
Subject
General Biochemistry, Genetics and Molecular Biology
Reference45 articles.
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2. The impact of replication stress on replication dynamics and DNA damage in vertebrate cells;Técher;Nat. Rev. Genet.,2017
3. Endogenous DNA Damage as a Source of Genomic Instability in Cancer;Tubbs;Cell,2017
4. Mechanisms of DNA Damage Tolerance: Post-Translational Regulation of PCNA;Leung;Genes,2018
5. RAD6-dependent DNA repair is linked to modification of PCNA by ubiquitin and SUMO;Hoege;Nature,2002
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1. Cell type specific suppression of hyper-recombination by human RAD18 is linked to PCNA K164 ubiquitination;2024-09-03
2. Clinical Validation of the Somatic FANCD2 Mutation (c.2022-5C>T) as a Novel Molecular Biomarker for Early Disease Progression in Chronic Myeloid Leukemia: A Case–Control Study;Hematology Reports;2024-07-08
3. Extracellular vesicles isolated from curcumin-medium weakened RKO cell proliferation and migration;Translational Cancer Research;2024-06
4. BRCA1/BARD1 ubiquitinates PCNA in unperturbed conditions to promote continuous DNA synthesis;Nature Communications;2024-05-20
5. Next-generation DNA sequencing identifies mutated FANCD2 Gene as a novel Biomarker for monitoring early disease progression and timely therapeutic interventions in advanced phase Chronic Myeloid Leukemia patients;2023-12-19
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