Reversal of DDK-Mediated MCM Phosphorylation by Rif1-PP1 Regulates Replication Initiation and Replisome Stability Independently of ATR/Chk1-Reference-Cited by-同舟云学术

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Reversal of DDK-Mediated MCM Phosphorylation by Rif1-PP1 Regulates Replication Initiation and Replisome Stability Independently of ATR/Chk1

Published:2017-03 Issue:10 Volume:18 Page:2508-2520
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Alver Robert C.,Chadha Gaganmeet Singh,Gillespie Peter J.,Blow J. Julian

Funder

Wellcome Trust

Cancer Research UK

Wellcome Trust Senior Investigator Award

Wellcome Trust Strategic Award

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference42 articles.

1. The Dbf4-Cdc7 kinase promotes Mcm2-7 ring opening to allow for single-stranded DNA extrusion and helicase assembly;Bruck;J. Biol. Chem.,2015

2. Mouse Rif1 is a key regulator of the replication-timing programme in mammalian cells;Cornacchia;EMBO J.,2012

3. An ATR- and Cdc7-dependent DNA damage checkpoint that inhibits initiation of DNA replication;Costanzo;Mol. Cell,2003

4. Protein phosphatase 1 recruitment by Rif1 regulates DNA replication origin firing by counteracting DDK activity;Davé;Cell Rep.,2014

5. Nuclear architecture organized by Rif1 underpins the replication-timing program;Foti;Mol. Cell,2016

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