The Inflammasome Drives GSDMD-Independent Secondary Pyroptosis and IL-1 Release in the Absence of Caspase-1 Protease Activity-Reference-Cited by-同舟云学术

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The Inflammasome Drives GSDMD-Independent Secondary Pyroptosis and IL-1 Release in the Absence of Caspase-1 Protease Activity

Published:2017-12 Issue:13 Volume:21 Page:3846-3859
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Schneider Katharina S.,Groß Christina J.,Dreier Roland F.,Saller Benedikt S.,Mishra Ritu,Gorka Oliver,Heilig Rosalie,Meunier Etienne,Dick Mathias S.,Ćiković Tamara,Sodenkamp Jan,Médard Guillaume,Naumann Ronald,Ruland Jürgen,Kuster Bernhard,Broz Petr,Groß Olaf

Funder

TUM Graduate School

Natural Sciences and Engineering Research Council of Canada

DFG

Swiss National Science Foundation

Bavarian Ministry of Sciences, Research and the Arts

European Research Council (ERC)

ERC

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference43 articles.

1. Inflammatory stimuli regulate caspase substrate profiles;Agard;Mol. Cell. Proteomics,2010

2. Proapoptotic chemotherapeutic drugs induce noncanonical processing and release of IL-1β via caspase-8 in dendritic cells;Antonopoulos;J. Immunol.,2013

3. Caspase-8 as an effector and regulator of NLRP3 inflammasome signaling;Antonopoulos;J. Biol. Chem.,2015

4. Salmonella induces macrophage death by caspase-1-dependent necrosis;Brennan;Mol. Microbiol.,2000

5. Inflammasomes: mechanism of assembly, regulation and signalling;Broz;Nat Rev Immunol.,2016

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