SF3B1 facilitates HIF1-signaling and promotes malignancy in pancreatic cancer

Author:

Simmler Patrik,Cortijo Cédric,Koch Lisa Maria,Galliker Patricia,Angori Silvia,Bolck Hella Anna,Mueller Christina,Vukolic Ana,Mirtschink Peter,Christinat Yann,Davidson Natalie R.,Lehmann Kjong-Van,Pellegrini Giovanni,Pauli Chantal,Lenggenhager Daniela,Guccini Ilaria,Ringel Till,Hirt Christian,Marquart Kim Fabiano,Schaefer Moritz,Rätsch Gunnar,Peter Matthias,Moch Holger,Stoffel Markus,Schwank GeraldORCID

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference45 articles.

1. Dysregulated splicing factor SF3B1 unveils a dual therapeutic vulnerability to target pancreatic cancer cells and cancer stem cells with an anti-splicing drug;Alors-Perez;J. Exp. Clin. Cancer Res.,2021

2. Cancer-associated SF3B1 mutations affect alternative splicing by promoting alternative branchpoint usage;Alsafadi;Nat. Commun.,2016

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4. SnakePipes: facilitating flexible, scalable and integrative epigenomic analysis;Bhardwaj;Bioinformatics,2019

5. Organoid models of human and mouse ductal pancreatic cancer;Boj;Cell,2015

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