Loss of Slc38a4 imprinting is a major cause of mouse placenta hyperplasia in somatic cell nuclear transferred embryos at late gestation
Author:
Funder
Howard Hughes Medical Institute
National Institutes of Health
Publisher
Elsevier BV
Subject
General Biochemistry, Genetics and Molecular Biology
Reference42 articles.
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1. Inhibition of Wnt activity improves peri-implantation development of somatic cell nuclear transfer embryos;National Science Review;2023-08-09
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3. Deciphering the placental abnormalities associated with somatic cell nuclear transfer at single-nucleus resolution;Protein & Cell;2023-05-19
4. Farrerol directly activates the deubiqutinase UCHL3 to promote DNA repair and reprogramming when mediated by somatic cell nuclear transfer;Nature Communications;2023-04-03
5. Noncanonical imprinting: intergenerational epigenetic inheritance mediated by Polycomb complexes;Current Opinion in Genetics & Development;2023-02
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