Loss of 4E-BP converts cerebellar long-term depression to long-term potentiation-Reference-Cited by-同舟云学术

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Loss of 4E-BP converts cerebellar long-term depression to long-term potentiation

Published:2022-06 Issue:10 Volume:39 Page:110911
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Saviuk Natasha,Chong Yumaine,Wang Peng^ORCID,Bermudez Sara,Zhao Zhe,Bhaskaran Arjun A.,Bowie Derek,Sonenberg Nahum,Cooper Ellis,Haghighi A. Pejmun

Funder

Buck Institute for Age Research

Canadian Institutes of Health Research

Research Institute for Aging, University of Waterloo

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference50 articles.

1. Isolation and characterization of PP2A holoenzymes containing FLAG-tagged B subunits;Adams;Methods Mol. Biol.,2007

2. Inhibition of group I metabotropic glutamate receptors reverses autistic-like phenotypes caused by deficiency of the translation repressor eIF4E binding protein 2;Aguilar-Valles;J. Neurosci.,2015

3. Deficient cerebellar long-term depression and impaired motor learning in mGluR1 mutant mice;Alba;Cell,1994

4. The translation repressor 4E-BP2 is critical for eIF4F complex formation, synaptic plasticity, and memory in the hippocampus;Banko;J. Neurosci.,2005

5. Behavioral alterations in mice lacking the translation repressor 4E-BP2;Banko;Neurobiol. Learn. Mem.,2007

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