ATM and SIRT6/SNF2H Mediate Transient H2AX Stabilization When DSBs Form by Blocking HUWE1 to Allow Efficient γH2AX Foci Formation-Reference-Cited by-同舟云学术

ATM and SIRT6/SNF2H Mediate Transient H2AX Stabilization When DSBs Form by Blocking HUWE1 to Allow Efficient γH2AX Foci Formation

Published:2015-12 Issue:12 Volume:13 Page:2728-2740
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Atsumi Yuko,Minakawa Yusuke,Ono Masaya,Dobashi Sachiko,Shinohe Keitaro,Shinohara Akira,Takeda Shunichi,Takagi Masatoshi,Takamatsu Nobuhiko,Nakagama Hitoshi,Teraoka Hirobumi,Yoshioka Ken-ichi

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference43 articles.

1. Onset of quiescence following p53 mediated down-regulation of H2AX in normal cells;Atsumi;PLoS ONE,2011

2. The Arf/p53 protein module, which induces apoptosis, down-regulates histone H2AX to allow normal cells to survive in the presence of anti-cancer drugs;Atsumi;J. Biol. Chem.,2013

3. H2AX may function as an anchor to hold broken chromosomal DNA ends in close proximity;Bassing;Cell Cycle,2004

4. Recognition of UbcH5c and the nucleosome by the Bmi1/Ring1b ubiquitin ligase complex;Bentley;EMBO J.,2011

5. Two-dimensional gel analysis of histones in acid extracts of nuclei, cells, and tissues;Bonner;Eur. J. Biochem.,1980

Cited by 87 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. New facets in the chromatin-based regulation of genome maintenance;DNA Repair;2024-08

2. An E3 ubiquitin ligase localization screen uncovers DTX2 as a novel ADP-ribosylation-dependent regulator of DNA double-strand break repair;Journal of Biological Chemistry;2024-08

3. E3 ligases: a ubiquitous link between DNA repair, DNA replication and human disease;Biochemical Journal;2024-07-10

4. Tom1p ubiquitin ligase structure, interaction with Spt6p, and function in maintaining normal transcript levels and the stability of chromatin in promoters;2024-07-08

5. Ki-67 is necessary during DNA replication for fork protection and genome stability;Genome Biology;2024-04-22