Long non-coding RNA CRNDE deteriorates intrauterine infection-induced neonatal brain injury
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference19 articles.
1. Perinatal brain injury: mechanisms, prevention, and outcomes;Novak;Clin. Perinatol.,2018
2. Dysregulation of neurogenesis by neuroinflammation: key differences in neurodevelopmental and neurological disorders;Fan;Neural Regen. Res.,2017
3. Effect of intrauterine infection on brain development and injury;Zhao;Int. J. Dev. Neurosci.,2013
4. Hypoxic postconditioning reduces microglial activation, astrocyte and caspase activity, and inflammatory markers after hypoxia-ischemia in the neonatal rat brain;Teo;Pediatr. Res.,2015
5. Differential microglial and astrocytic response to bacterial and viral infection in the developing hippocampus of neonatal rats;Patro;Indian J. Exp. Biol.,2013
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1. Circ_19038 and lnc-AK016022 synergistically regulate Sirt1 to promote remyelination and alleviate white matter injury in preterm mice;Archives of Biochemistry and Biophysics;2024-10
2. SIRT1 inhibits Smad3 acetylation by reducing Lnc CRNDE transcription and inhibits BV2 cell overactivation to promote SCI neurological repair;2023-09-20
3. The expression profile analysis of high-throughput lncRNA sequencing in preterm brain tissue induced by intrauterine infection/inflammation;2023-09-12
4. Non-Coding RNA in Microglia Activation and Neuroinflammation in Alzheimer’s Disease;Journal of Inflammation Research;2023-09
5. LncRNA CRNDE binds hnRNPA1 to facilitate carbon monoxide poisoning-induced delayed encephalopathy via inhibiting UCHL5-mediated SMO deubiquitination;Metabolic Brain Disease;2023-01-17
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