Anti-IL-17A blocking antibody reduces cyclosporin A-induced relapse in experimental autoimmune encephalomyelitis mice-Reference-Cited by-同舟云学术

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Anti-IL-17A blocking antibody reduces cyclosporin A-induced relapse in experimental autoimmune encephalomyelitis mice

Published:2016-12 Issue: Volume:8 Page:139-145
ISSN:2405-5808
Container-title:Biochemistry and Biophysics Reports
language:en
Short-container-title:Biochemistry and Biophysics Reports

Author:

Saitoh Kodai,Kon Shigeyuki,Nakatsuru Takuya,Inui Kyosuke,Ihara Takeru,Matsumoto Naoki,Kitai Yuichi,Muromoto Ryuta,Matsuda Tadashi

Funder

JSPS KAKENHI

The Fugaku Trust for Medical Research

Publisher

Elsevier BV

Subject

Biochemistry,Biophysics

Reference35 articles.

1. Mechanisms of action of cyclosporine;Matsuda;Immunopharmacology,2000

2. Cyclosporine, tolerance, and autoimmunity;Prud'homme;Clin. Immunol. Immunopathol.,1993

3. The use of cyclosporine in dermatology: (Part I);Amor;J. Am. Acad. Dermatol.,2010

4. Induction of apoptosis and modulation of activation and effector function in T cells by immunosuppressive drugs;Strauss;Clin. Exp. Immunol.,2002

5. Concentrations of cyclosporin A and FK506 that inhibit IL-2 induction in human T cells do not affect TGF-beta1 biosynthesis, whereas higher doses of cyclosporin A trigger apoptosis and release of preformed TGF-beta1;Minguillon;J. Leukoc. Biol.,2005

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