Lack of association of ovariectomy-induced obesity with overeating and the reduction of physical activities-Reference-Cited by-同舟云学术

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Lack of association of ovariectomy-induced obesity with overeating and the reduction of physical activities

Published:2019-12 Issue: Volume:20 Page:100671
ISSN:2405-5808
Container-title:Biochemistry and Biophysics Reports
language:en
Short-container-title:Biochemistry and Biophysics Reports

Author:

Nishio Eiji,Hayashi Takanori,Nakatani Masashi,Aida Noriko,Suda Risa,Fujii Takuma,Wakatsuki Toru,Honda Shinichiro,Harada Nobuhiro,Shimono Yohei

Funder

Takeda Science Foundation

Fujita Health University, Japan

Publisher

Elsevier BV

Subject

Biochemistry,Biophysics

Reference20 articles.

1. Global burden of obesity in 2005 and projections to 2030;Kelly;Int. J. Obes.,2008

2. Health effects of overweight and obesity in 195 countries over 25 Years;Afshin;N. Engl. J. Med.,2017

3. The relation of body mass index and abdominal adiposity with dyslipidemia in 27 general populations of the WHO MONICA Project;Wietlisbach;Nutr. Metab. Cardiovasc. Dis.,2013

4. Type 2 diabetes from pediatric to geriatric age: analysis of gender and obesity among 120,183 patients from the German/Austrian DPV database;Awa;Eur. J. Endocrinol.,2012

5. Involvement of epidermal growth factor in inducing obesity in ovariectomized mice;Kurachi;Am. J. Physiol.,1993

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1. Strength Training Protects High-Fat-Fed Ovariectomized Mice against Insulin Resistance and Hepatic Steatosis;International Journal of Molecular Sciences;2024-05-07

2. Effect of calorie restriction on redox status during chemically induced estropause in female mice;GeroScience;2023-10-19

3. Feeding neurons integrate metabolic and reproductive states in mice;iScience;2023-10

4. Role of Mast-Cell-Derived RANKL in Ovariectomy-Induced Bone Loss in Mice;International Journal of Molecular Sciences;2023-05-23

5. Histological Analysis of Hepatic Steatosis, Inflammation, and Fibrosis in Ascorbic Acid-Treated Ovariectomized Mice;Biomedical Science Letters;2022-06-30

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