GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression-Reference-Cited by-同舟云学术

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GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression

Published:2021-12 Issue: Volume:28 Page:101145
ISSN:2405-5808
Container-title:Biochemistry and Biophysics Reports
language:en
Short-container-title:Biochemistry and Biophysics Reports

Author:

Lian Zhiwen,Ke Guibao,Zhang Hong,Dou Caoshuai,Chen Xueqin,Li Bohou,Zhang Fengxia,Wen Shichun,Wu Qiong,Xia Yubin,Jiang Nan,Li Zhuo,Li Sijia,Zhao Xingchen,Ma Jianchao,Lin Ting,Wen Feng,Xu Lixia,Li Zhilian,Liang Huabang,Dong Wei,Chen Yuanhan,Li Ruizhao,Ye Zhiming,Wang Wenjian,Liang Xinling,Shi Wei,Zhang Li,Liu Shuangxin

Publisher

Elsevier BV

Subject

Biochemistry,Biophysics

Reference30 articles.

1. LPS and PAN-induced podocyte injury in an in vitro model of minimal change disease: changes in TLR profile;Srivastava;J Cell Commun Signal,2013

2. Lipopolysaccharide-induced podocyte injury is mediated by suppression of autophagy;Tan;Mol. Med. Rep.,2016

3. Podocyte injury induces nuclear translocation of WTIP via microtubule-dependent transport;Kim;J. Biol. Chem.,2010

4. Secondary focal segmental glomerulosclerosis: from podocyte injury to glomerulosclerosis;Kim;BioMed Res. Int.,2016

5. The role of podocytes in glomerular pathobiology;Asanuma;Clin. Exp. Nephrol.,2003

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Transcriptomic profile of human iPSC-derived podocyte-like cells exposed to a panel of xenobiotics;Toxicology in Vitro;2024-05

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