PPP2R4 dysfunction promotes KRAS-mutant lung adenocarcinoma development and mediates opposite responses to MEK and mTOR inhibition-Reference-Cited by-同舟云学术

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PPP2R4 dysfunction promotes KRAS-mutant lung adenocarcinoma development and mediates opposite responses to MEK and mTOR inhibition

Published:2021-11 Issue: Volume:520 Page:57-67
ISSN:0304-3835
Container-title:Cancer Letters
language:en
Short-container-title:Cancer Letters

Author:

Meeusen Bob,Cortesi Emanuela Elsa^ORCID,Domènech Omella Judit,Sablina Anna,Ventura Juan-Jose^ORCID,Janssens Veerle^ORCID

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference47 articles.

1. Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries;Bray;Ca - Cancer J. Clin.,2018

2. KRAS-Mutant non-small cell lung cancer: from biology to therapy;Ferrer;Lung Cancer,2018

3. Targeting the MAPK pathway in KRAS-driven tumors;Drosten;Cancer Cell,2020

4. Protein phosphatase 2A: a highly regulated family of serine/threonine phosphatases implicated in cell growth and signalling;Janssens;Biochem. J.,2001

5. Physiologic functions of PP2A: lessons from genetically modified mice;Reynhout;Biochim. Biophys. Acta Mol. Cell Res.,2019

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