Mechanisms used by cancer cells to tolerate drug-induced replication stress-Reference-Cited by-同舟云学术

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Mechanisms used by cancer cells to tolerate drug-induced replication stress

Published:2022-09 Issue: Volume:544 Page:215804
ISSN:0304-3835
Container-title:Cancer Letters
language:en
Short-container-title:Cancer Letters

Author:

Segeren Hendrika A.,Westendorp Bart^ORCID

Funder

KWF Kankerbestrijding

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference109 articles.

1. DNA replication stress as a hallmark of cancer;Macheret;Annu. Rev. Pathol.,2015

2. Beyond kinases: targeting replication stress proteins in cancer therapy;Baillie;Trends Cancer,2021

3. Increased replication initiation and conflicts with transcription underlie Cyclin E-induced replication stress;Jones;Oncogene,2013

4. Cdc45 is a critical effector of myc-dependent DNA replication stress;Srinivasan;Cell Rep.,2013

5. Intragenic origins due to short G1 phases underlie oncogene-induced DNA replication stress;Macheret;Nature,2018

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1. Replication stress as a driver of cellular senescence and aging;Communications Biology;2024-05-22

2. The KRAS, ATR and CHEK1 expression levels in endometrial cancer are the risk factors predicting recurrence;PLOS ONE;2024-04-26

3. Ceramide kinase-mediated C1P metabolism attenuates acute liver injury by inhibiting the interaction between KEAP1 and NRF2;Experimental & Molecular Medicine;2024-04-01

4. The Novel ATR Inhibitor Tuvusertib (M1774) Induces Replication Protein Overexpression and Broad Synergy with DNA-targeted Anticancer Drugs;Molecular Cancer Therapeutics;2024-03-11

5. Synergistic lethality between auranofin-induced oxidative DNA damage and ATR inhibition in cancer cells;Life Sciences;2023-11

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