A mutual activation loop between the Ca2+-activated chloride channel TMEM16A and EGFR/STAT3 signaling promotes breast cancer tumorigenesis-Reference-Cited by-同舟云学术

A mutual activation loop between the Ca2+-activated chloride channel TMEM16A and EGFR/STAT3 signaling promotes breast cancer tumorigenesis

Published:2019-07 Issue: Volume:455 Page:48-59
ISSN:0304-3835
Container-title:Cancer Letters
language:en
Short-container-title:Cancer Letters

Author:

Wang Hui,Yao Fan,Luo Shuya,Ma Ke,Liu Mei,Bai Lichuan,Chen Si,Song Chang,Wang Tianyu,Du Qiang,Wu Huizhe,Wei Minjie,Fang Yue,Xiao Qinghuan^ORCID

Funder

National Natural Science Foundation of China

Liaoning Pandeng Scholar

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference32 articles.

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4. Cell-specific regulation of proliferation by Ano1/TMEM16A in breast cancer with different ER, PR, and HER2 status;Wu;Oncotarget,2017

5. TMEM16A induces MAPK and contributes directly to tumorigenesis and cancer progression;Duvvuri;Cancer Res.,2012

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