mTORC2 contributes to the metabolic reprogramming in EGFR tyrosine-kinase inhibitor resistant cells in non-small cell lung cancer-Reference-Cited by-同舟云学术

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mTORC2 contributes to the metabolic reprogramming in EGFR tyrosine-kinase inhibitor resistant cells in non-small cell lung cancer

Published:2018-10 Issue: Volume:434 Page:152-159
ISSN:0304-3835
Container-title:Cancer Letters
language:en
Short-container-title:Cancer Letters

Author:

Chiang Chun-Te,Demetriou Alexandra N.,Ung Nolan,Choudhury Niharika,Ghaffarian Kimya,Ruderman Daniel L.,Mumenthaler Shannon M.

Funder

National Cancer Institute

Physical Sciences-Oncology Center (PS-OC)

University of Southern California

USC Medical Faculty Women's Association

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference45 articles.

1. Cancer Statistics, 2018, CA: a Cancer Journal for Clinicians;Siegel,2018

2. Acquired resistance of lung adenocarcinomas to gefitinib or erlotinib is associated with a second mutation in the EGFR kinase domain;Pao;PLoS Medicine,2005

3. The quest to overcome resistance to EGFR-targeted therapies in cancer;Chong;Nat. Med.,2013

4. Treatment of nonsmall cell lung cancer: overcoming the resistance to epidermal growth factor receptor inhibitors;Carter;Curr. Opin. Oncol.,2012

5. Acquired EGFR C797S mutation mediates resistance to AZD9291 in non-small cell lung cancer harboring EGFR T790M;Thress;Nat. Med.,2015

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