The risk of cardiovascular events with increased apolipoprotein CIII: A systematic review and meta-analysis

Author:

Wyler von Ballmoos Moritz C.,Haring Bernhard,Sacks Frank M.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Nutrition and Dietetics,Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference46 articles.

1. Mechanisms of inhibition by apolipoprotein C of apolipoprotein E-dependent cellular metabolism of human triglyceride-rich lipoproteins through the low density lipoprotein receptor pathway;Sehayek;J Biol Chem,1991

2. The estradiol-stimulated lipoprotein receptor of rat liver. A binding site that membrane mediates the uptake of rat lipoproteins containing apoproteins B and E;Windler;J Biol Chem,1980

3. Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles;Aalto-Setala;J Clin Invest,1992

4. Further characterization of the metabolic properties of triglyceride-rich lipoproteins from human and mouse apoC-III transgenic mice;Aalto-Setala;J Lipid Res,1996

5. Overexpression of human apolipoprotein C-III in transgenic mice results in an accumulation of apolipoprotein B48 remnants that is corrected by excess apolipoprotein E;de Silva;J Biol Chem,1994

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