Co-exposure of arsenic and polystyrene-nanoplastics induced kidney injury by disrupting mitochondrial homeostasis and mtROS-mediated ferritinophagy and ferroptosis-Reference-Cited by-同舟云学术

Co-exposure of arsenic and polystyrene-nanoplastics induced kidney injury by disrupting mitochondrial homeostasis and mtROS-mediated ferritinophagy and ferroptosis

Published:2024-05 Issue: Volume:201 Page:105904
ISSN:0048-3575
Container-title:Pesticide Biochemistry and Physiology
language:en
Short-container-title:Pesticide Biochemistry and Physiology

Author:

Zhong Gaolong,Qiao Baoxin,He Ying,Liu Haiyan,Hong Panjing,Rao Gan,Tang Lixuan,Tang Zhaoxin,Hu Lianmei

Funder

Nanning Science and Technology Base Project

Publisher

Elsevier BV

Reference56 articles.

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3. Nephrotoxicity and renal pathophysiology: a contemporary perspective;Barnett;Toxicol. Sci.,2018

4. Mitochondria targeting by environmental stressors: implications for redox cellular signaling;Blajszczak;Toxicology,2017

5. The nephrotoxic potential of polystyrene microplastics at realistic environmental concentrations;Chen;J. Hazard. Mater.,2022

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1. Arsenic trioxide induces innate immune response and inflammatory response in chicken liver via cGAS-STING/NF-κB pathway;Comparative Biochemistry and Physiology Part C: Toxicology & Pharmacology;2024-12

2. Naringenin counteracts LPS-induced inflammation and immune deficits in chicken thymus by alleviating mtROS/ferroptosis levels;Poultry Science;2024-11

3. Arsenic-induced mtDNA release promotes inflammatory responses through cGAS-STING signaling in chicken hepatocytes;Pesticide Biochemistry and Physiology;2024-09

4. Ferroptosis: a novel mechanism of cell death in ophthalmic conditions;Frontiers in Immunology;2024-06-27