Eicosanoids in sickle cell disease: potential relevance of 12(s)-hydroxy-5,8,10,14-eicosatetraenoic acid to the pathophysiology of vaso-occlusion

Author:

Setty B.N.Yamaja,Chen Dechun,O'Neal Pat,Littrell James B.,Grossman Mark H.,Stuart Marie J.

Publisher

Elsevier BV

Subject

General Medicine,Pathology and Forensic Medicine

Reference42 articles.

1. Hydroxyeicosatetraenoicacids (HETEs);Spector;Prog Lipid Res,1988

2. Brain microvessels produce 12-hydroxyeicosatetraenoic acid;Moore;J Neurochem,1989

3. Bidirectional control of membrane expression and/or activation of the tumor cell IRGpIIb/IIIa receptor and tumor cell adhesion by lipoxygenase products of arachidonic acid and linoleic acid;Grossi;Cancer Res,1989

4. The lipoxygenase metabolite 12(S)-HETE induces a cytoskeleton-dependent increase in surface expression of integrin αIIbβ3 on melanoma cells;Chopra;Int J Cancer,1991

5. Protein Kinase C-dependent effects of 12(S)-HETE on endothelial cell vitronectin receptor and fibronectin receptor;Tang;J Cell Biol,1993

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