Involvement of antipain-sensitive protease activity in the interferon-β-induced UV-refractoriness of Cockayne syndrome fibroblasts
Author:
Publisher
Elsevier BV
Subject
Health, Toxicology and Mutagenesis,Genetics,Molecular Biology
Reference23 articles.
1. Excision repair in ataxia telangiectasia, Fanconi's anemia, Cockayne syndrome, and Bloom's syndrome after treatment with ultraviolet radiation and N-acetoxy-2-acethylaminofluorene;Ahmed;Biochim. Biophys. Acta,1978
2. Cross-sensitivity of certain xeroderma pigmentosum and Cockayne syndrome fibroblast strains to both ionizing radiation and ultraviolet light;Chan;Mol. Gen. Genet.,1981
3. Repair in ribosomal RNA genes is deficient in xeroderma pigmentosum group C and Cockayne's syndrome cells;Christians;Mutations Res.,1994
4. Dwarfism with retinal atrophy and deafness;Cockayne;Arch. Dis. Child,1936
5. Unusual sensitivity of two Cockayne's syndrome cell strains to both UV and γ-irradiation;Deschavanne;Mutation Res.,1981
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1. Molecular Regulation of UV-Induced DNA Repair;Photochemistry and Photobiology;2015-01-14
2. Mechanisms of human cell resistance to mutagens;Biology Bulletin Reviews;2011-11
3. Mechanisms of human cell protection associated with genetic polymorphism;Russian Journal of Genetics;2005-04
4. Studies on p53 and Bax protein expression in Cockayne syndrome cells after UV irradiation and interferon-? treatment;Cell Biochemistry and Function;2001
5. Protease activation following UV irradiation is linked to hypomutability in human cells selected for resistance to combination of UV and antipain;Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis;1998-07
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