Poly(ADP-ribose) metabolism appears normal in EM9, a mutagen-sensitive mutant of CHO cells
Author:
Publisher
Elsevier BV
Subject
Health, Toxicology and Mutagenesis,Genetics,Molecular Biology
Reference20 articles.
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1. The high rate of endoreduplication in the repair deficient CHO mutant EM9 parallels a reduced level of methylated deoxycytidine in DNA;Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis;2008-09-26
2. XRCC1 and DNA polymerase β in cellular protection against cytotoxic DNA single-strand breaks;Cell Research;2008-01
3. MNNG-induced Cell Death Is Controlled by Interactions between PARP-1, Poly(ADP-ribose) Glycohydrolase, and XRCC1;Journal of Biological Chemistry;2006-11
4. The Novel Poly(ADP-Ribose) Polymerase Inhibitor, AG14361, Sensitizes Cells to Topoisomerase I Poisons by Increasing the Persistence of DNA Strand Breaks;Clinical Cancer Research;2005-12-01
5. XRCC1 keeps DNA from getting stranded;Mutation Research/DNA Repair;2000-02
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