Fetal anomalies produced subsequent to treatment of zygotes with ethylene oxide or ethyl methanesulfonate are not likely due to the usual genetic causes
Author:
Publisher
Elsevier BV
Subject
Health, Toxicology and Mutagenesis,Genetics,Molecular Biology
Reference18 articles.
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2. Fate and metabolism of some mutagenic alkylating agents in the mouse, I. Ethyl methylsulfonate and methyl methanesulfonate at sublethal dose in hybrid males;Cumming;Mutation Res.,1970
3. Dominant-lethal mutations and heritable translocations in mice;Generoso,1984
4. Exposure of female mice to ethylene oxide within hours after mating leads to fetal malformation and death;Generoso;Mutation Res.,1987
5. Mutagen-induced fetal anomalies and death following treatment of females within hours after mating;Generoso;Mutation Res.,1988
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2. Developmental defects and genomic instability after x-irradiation of wild-type and genetically modified mouse pre-implantation and early post-implantation embryos;Journal of Radiological Protection;2012-10-03
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4. The all-or-none phenomenon revisited;Birth Defects Research Part A: Clinical and Molecular Teratology;2012-07-17
5. Literature review on the genotoxicity, reproductive toxicity, and carcinogenicity of ethyl methanesulfonate;Toxicology Letters;2009-11-12
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