A cell-cycle stage-related chromosomal X-ray hypersensitivity in larval neuroblast of Drosophila mei-9 and mei-41 mutants suggesting defecting DNA double-strand break repair-Reference-Cited by-同舟云学术

A cell-cycle stage-related chromosomal X-ray hypersensitivity in larval neuroblast of Drosophila mei-9 and mei-41 mutants suggesting defecting DNA double-strand break repair

Published:1989-03 Issue:1 Volume:211 Page:111-124
ISSN:0027-5107
Container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
language:en
Short-container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis

Author:

Schweizer Pascal M.

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics,Molecular Biology

Reference47 articles.

1. Do mammalian cells leave a fraction of DNA breaks unrepaired after ionizing radiation?;Ahnström,1982

2. Genetic control of meiotic recombination and somatic DNA metabolism in Drosophila melanogaster;Baker,1976

3. Utilization during meiotic cell division of loci controlling meiotic recombination and disfunction in Drosophila melanogaster;Baker;Genetics,1978

4. Effects of recombination-deficient and repair-deficient loci on meiotic chromosome behaviour in Drosophila melanogaster;Baker,1980

5. Relationship of DNA lesions and their repair to chromosomal aberration production;Bender,1980

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1. White-ivory assay ofDrosophila melanogaster under deficient repair conditions;Environmental and Molecular Mutagenesis;1998

2. Molecular cloning of mei-41, a gene that influences both somatic and germline chromosome metabolism of Drosophila melanogaster;Molecular and General Genetics MGG;1995-03

3. Radiation and transposon-induced genetic damage in Drosophila melanogaster: X-ray dose-response and synergism with DNA-repair deficiency;Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis;1992-05

4. P transposition in Drosophila provides a new tool for analyzing postreplication repair and double-strand break repair;Mutation Research/DNA Repair;1991-07

5. Adaptive Response to Dominant Lethality of Mature (Class A) and Immature (Class B) Oocytes ofD. Melanogasterto Low Doses of Ionizing Radiation: Effects in Repair-proficient (yw) and Repair-deficient Strains (Mei 41D5and Mus 302D1);International Journal of Radiation Biology;1991-01