Nitroglycerin (exogenous nitric oxide) substitutes for endothelium-derived nitric oxide in potentiating vasorelaxations and cyclic AMP elevations induced by calcitonin gene-related peptide (CGRP) in rat aorta

Author:

Fiscus R.R.,Hao H.,Wang X.,Arden W.A.,Diana J.N.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrine and Autonomic Systems,Neurology,Endocrinology,General Medicine

Reference34 articles.

1. Molecular mechanisms of endothelium-mediated vasodilation;Fiscus;Semin. Thromb. Hemost.,1988

2. Calcitonin gene-related peptide (CGRP) causes endothelium-dependent cyclic AMP, cyclic GMP and vasorelaxant responses in rat abdominal aorta;Wang;Neuropeptides,1991

3. Calcitonin gene-related peptide (CGRP)-induced cyclic AMP, cyclic GMP and vasorelaxant responses in rat thoracic aorta are antagonized by blockers of endothelium-derived relaxant factor (EDRF);Fiscus;Neuropeptides,1991

4. Mechanism of vascular relaxation by the calcitonin gene-related peptide;Marshall,1992

5. A pharmacological profile of the endothelium-derived relaxant factor released by calcitonin gene-related peptide in rat aorta;Gray,1992

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