Increased numbers of substance P-containing sensory neurons in a rat strain with a genetic neurotrophic defect
Author:
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Endocrine and Autonomic Systems,Neurology,Endocrinology,General Medicine
Reference22 articles.
1. Cutaneous stimuli releasing immunoreactive substance P in the dorsal horn of the cat;Duggan;Brain Res.,1988
2. Electrophysiological and neurochemical evidence for substance P as a transmitter of primary sensory neurons;Otsuka,1977
3. Antidromic vasodilation and neurogenic inflammation;Chahl,1991
4. Local effector functions of capsaicin-sensitive sensory nerve endings: involvement of tachykinins, calcitonin gene-related peptide and other neuropeptides;Holzer;Neuroscience,1988
5. The origin and possible significance of substance P immunoreactive networks in the prevertebral ganglia and related structures in the guinea-pig;Matthews;Phil. Trans. Roy. Soc. B,1984
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. The role of calcium channels in substance P-induced contractile response in the rat iris;Peptides;2003-12
2. Reduced inflammation in genetically hypertensive rat airways is associated with reduced tachykinin NK1 receptor numbers;European Journal of Pharmacology;2000-07
3. Decreased vascular permeability response to substance P in airways of genetically hypertensive rats;British Journal of Pharmacology;1999-02
4. Decreased developmental cell death in sympathetic and spinal sensory nervous systems of the Kyoto spontaneously hypertensive rat;Journal of Hypertension;1996-09
5. Neurokinin A and substance P vary independently in different regions of rat sensory neurons;Neuropeptides;1995-04
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