In vivo decrease of adenylate cyclase responsiveness to isoproterenol in the spontaneously hypertensive rat.
Author:
Publisher
Elsevier BV
Subject
Pharmacology,Biochemistry
Reference11 articles.
1. Altered function of adenylate cyclase in the myocardium of the spontaneously hypertensive rat
2. Cyclic adenosine monophosphate and vascular reactivity in spontaneously hypertensive rats
3. Ontogenetic development of isoproterenol subsensitivity of myocardial adenylate cyclase and β-adrenergic receptors in spontaneously hypertensive rats
4. Reduced number of β-adrenergic receptors in the myocardium of spontaneously hypertensive rats
5. Decreased number of beta-adrenergic receptors in hypertensive vessels
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Neurochemical coupled actions of transmitters in the microvasculature of the brain;Neuroscience & Biobehavioral Reviews;1986-06
2. Mechanisms of Smooth Muscle Relaxation;Calcium and Contractility;1985
3. Age-related changes of in vivo β-adrenergic responsiveness in normotensive and spontaneously hypertensive rats;European Journal of Pharmacology;1984-06
4. ß-Adrenergic Receptors;Handbook of Neurochemistry;1984
5. Ageing as a factor governing the effect of isoproterenol on the cyclic AMP level in isolated aorta from spontaneously hypertensive and normotensive rats;European Journal of Pharmacology;1983-01
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