The proton pump inhibitor, E3810, binds to the N-terminal half of the α-subunit of gastric H+,K+-ATPase-Reference-Cited by-同舟云学术

The proton pump inhibitor, E3810, binds to the N-terminal half of the α-subunit of gastric H+,K+-ATPase

Published:1995-06 Issue:12 Volume:49 Page:1729-1734
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Morii Magotoshi,Hamatani Kiyomi,Takeguchi Noriaki

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference28 articles.

1. The potency of substituted benzimidazoles such as E3810, omeprazole, Ro 18-5364 to inhibit gastric H+,K+-ATPase is correlated with the rate of acid-activation of the inhibitor;Morii;Biochem Pharmacol,1990

2. Inhibitions of acid secretion by E3810 and omeprazole, and their reversal by glutathione;Fujisaki;Biochem Pharmacol,1991

3. The mechanism of action of the gastric acid secretion inhibitor omeprazole;Lindberg;J Med Chem,1986

4. Acid activation of (H+-K+)-ATPase inhibiting 2-(2-pyridylmethylsulphinyl benzimidazoles: isolation and characterization of the thiophilic ‘Active principle’ and its reactions;Figera;J Chem Soc Chem Commun,1986

5. Studies on the mechanism of action of the omeprazole-derived cyclic sulphenamide;Beil;Biochem Pharmacol,1988

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