In vivo and in vitro effects of nafazatrom (Bay g 6575), an antithrombotic compound, on arachidonic acid metabolism in platelets and vascular tissue
Author:
Publisher
Elsevier BV
Subject
Pharmacology,Biochemistry
Reference53 articles.
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3. Regulation of arachidonate-induced platelet aggregation by the lipoxygenase product, 12-hydroperoxyeicosatetraenoic acid
4. A lipid peroxide inhibits the enzyme in blood vessel microsomes that generates from prostaglandin endoperoxides the substance (prostaglandin X) which prevents platelet aggregation
5. Prostacyclin (prostaglandin I2, PGI2) inhibits platelet adhesion and thrombus formation on subendothelium
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1. Effects of Leukotriene Synthesis Inhibition on Acute Liver Damage Induced by Carbon Tetrachloride;Pharmacology;1993
2. Inhibition by REV-5901 of leukotriene release from guinea-pig and human lung tissue in vitro;Biochemical Pharmacology;1989-12
3. Investigations on the role of arachidonic acid metabolism pathways in the antithrombotic activity of nafazatrom and molsidomine;Thrombosis Research;1988-06
4. Effect of nafazatrom and indomethacin on pulmonary removal of prostaglandin E1 after endotoxin in rabbits;British Journal of Pharmacology;1987-08
5. Stimulation of vascular prostacyclin by SKF525-A (proadifen) and related compounds;Biochemical Pharmacology;1987-05
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