The fate of N1′-methanesulphonyl-N4′-(9-acridinyl) -3′ -methoxy-2′,5′ -cyclohexadiene-1′,4′-diimine (m-aqdi), the primary oxidative metabolite of amsacrine, in transformed chinese hamster fibroblasts-Reference-Cited by-同舟云学术

The fate of N1′-methanesulphonyl-N4′-(9-acridinyl) -3′ -methoxy-2′,5′ -cyclohexadiene-1′,4′-diimine (m-aqdi), the primary oxidative metabolite of amsacrine, in transformed chinese hamster fibroblasts

Published:1990-05 Issue:9 Volume:39 Page:1411-1421
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Robbie Maxine A.,Palmer Brian D.,Denny William A.,Wilson William R.

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference30 articles.

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3. Intercalative antitumor drugs interfere with the breakage-reunion reaction in mammalian DNA topoisomerase II.;Tewey;J Biol Chem,1984

4. DNA topoisomerase II: a primer on the enzyme and its unique role as a multidrug target in cancer chemotherapy;Glisson;Pharmacol Ther,1987

5. Comparison of the mutagenic and clastogenic activity of amsacrine and other DNA-intercalating drugs in cultured V79 Chinese hamstercells;Wilson;Cancer Res,1984

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