Heterozygous Inactivation of the Vinculin Gene Predisposes to Stress-Induced Cardiomyopathy-Reference-Cited by-同舟云学术

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Heterozygous Inactivation of the Vinculin Gene Predisposes to Stress-Induced Cardiomyopathy

Published:2004-09 Issue:3 Volume:165 Page:1033-1044
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Zemljic-Harpf Alice E.,Ponrartana Sornya,Avalos Roy T.,Jordan Maria C.,Roos Kenneth P.,Dalton Nancy D.,Phan Vinh Q.,Adamson Eileen D.,Ross Robert S.

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference55 articles.

1. Differential actin organization by vinculin isoforms: implications for cell type-specific microfilament anchorage;Rudiger;FEBS Lett,1998

2. Signal transduction from the extracellular matrix—a role for the focal adhesion protein-tyrosine kinase FAK;Schlaepfer;Cell Struct Funct,1996

3. Identification of sequences required for the efficient localization of the focal adhesion kinase, pp125FAK, to cellular focal adhesions;Hildebrand;J Cell Biol,1993

4. The vinculin/sarcomeric-α-actinin/α-actin nexus in cultured cardiac myocytes;Lu;J Cell Biol,1992

5. Vinculin and α-actinin: interaction with actin and effect on microfilament network formation;Jockusch;Cold Spring Harbor Symp Quant Biol,1982

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