Recording oral activity in rats reveals a long-lasting subsensitivity to haloperidol as a function of duration of previous haloperidol treatment
Author:
Publisher
Elsevier BV
Subject
Behavioral Neuroscience,Biological Psychiatry,Clinical Biochemistry,Pharmacology,Toxicology,Biochemistry
Reference14 articles.
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1. New Models of Frontal-Subcortical Skeletomotor Circuit Pathology in Tardive Dyskinesia;The Neuroscientist;2006-06
2. The vacuous chewing movement (VCM) model of tardive dyskinesia revisited: is there a relationship to dopamine D2 receptor occupancy?;Neuroscience & Biobehavioral Reviews;2002-05
3. Chronic haloperidol potentiates stimulated glutamate release in caudate putamen, but not prefrontal cortex;NeuroReport;1995-09
4. Dose-dependent differences in the development of reserpine-induced oral dyskinesia in rats: support for a model of tardive dyskinesia;Psychopharmacology;1994-09
5. Changes in striatal dopamine release and metabolism during and after subchronic haloperidol administration in rats;Neuroscience Letters;1992-08
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