Effect of acute ethanol exposure during pregnancy on dentate gyrus synaptic plasticity in 45-day-old rats
Author:
Publisher
Elsevier BV
Subject
Behavioral Neuroscience,Biological Psychiatry,Clinical Biochemistry,Pharmacology,Toxicology,Biochemistry
Reference51 articles.
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3. Deficits on a spatial navigation task following prenatal exposure to ethanol;Blanchard;Neurotoxicol. Teratol.,1987
4. Where is the locus of long-term potentiation?;Bliss,1984
5. Long-lasting potentiation of synaptic transmission in dentate area of the anesthetized rabbit following stimulation of the perforant path;Bliss;J. Physiol. (Lond.),1973
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1. Increase of KCC2 in hippocampal synaptic plasticity disturbances after perinatal ethanol exposure;Addiction Biology;2016-10-25
2. Aberrant NMDA-dependent LTD after perinatal ethanol exposure in young adult rat hippocampus;Hippocampus;2015-01-23
3. Development of multi-route physiologically-based pharmacokinetic models for ethanol in the adult, pregnant, and neonatal rat;Inhalation Toxicology;2012-09
4. Neonatal ethanol exposure reduces AMPA but not NMDA receptor levels in the rat neocortex;Developmental Brain Research;2002-05
5. A longitudinal study of the effects of prenatal ethanol exposure on neuronal acquisition and death in the principal sensory nucleus of the trigeminal nerve: interaction with changes induced by transection of the infraorbital nerve;Journal of Neurocytology;1999
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