The relationship between oral dyskinesias produced by long-term haloperidol treatment, the density of striatal preproenkephalin messenger RNA and enkephalin peptide, and the number of striatal neurons expressing preproenkephalin messenger RNA in rats-Reference-Cited by-同舟云学术

The relationship between oral dyskinesias produced by long-term haloperidol treatment, the density of striatal preproenkephalin messenger RNA and enkephalin peptide, and the number of striatal neurons expressing preproenkephalin messenger RNA in rats

Published:1999-01 Issue:1 Volume:88 Page:27-35
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Andreassen O.A.,Finsen B.,Østergaard K.,Sørensen J.C.,West M.J.,Jørgensen H.A.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference49 articles.

1. American Psychiatric Association Task Force on Tardive Dyskinesia (1992) Tardive Dyskinesia: A Task Force Report of the American Psychiatric Association. APA Press; Washington, DC.

2. GM1 ganglioside attenuates the development of vacuous chewing movements induced by long-term haloperidol treatment of rats;Andreassen;Psychopharmacology,1994

3. The mitochondrial toxin 3-nitropropionic acid induces vacuous chewing movements in rats. Implications for tardive dyskinesia?;Andreassen;Psychopharmacology,1995

4. Inhibition by memantine of the development of persistent oral dyskinesias induced by long-term haloperidol treatment of rats;Andreassen;Br. J. Pharmac.,1996

5. Oral dyskinesias and morphological changes in striatum in rats during long-term haloperidol treatment;Andreassen;Soc. Neurosci. Abstr.,1997

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