Degeneration of neurons and glia in the Niemann–Pick C mouse is unrelated to the low-density lipoprotein receptor

Author:

German D.C,Quintero E.M,Liang C.-L,Xie C,Dietschy J.M

Publisher

Elsevier BV

Subject

General Neuroscience

Reference32 articles.

1. Type-C Niemann–Pick disease: low density lipoprotein uptake is associated with premature cholesterol accumulation in the Golgi complex and excessive cholesterol storage in lysosomes;Blanchette-Mackie;Proc. Natl. Acad. Sci. USA,1988

2. Brady, R.O., 1983. The metabolic basis of inherited disease. In: Stanbury, J.B., Wyngaarden, J.B., Fredrickson, D.S., Goldstein, J.L., Brown, M.S. (Eds.), Sphingomyelin Lipidosis: Niemann–Pick Disease. McGraw Hill, New York, pp. 834–841.

3. Niemann–Pick C1 disease gene: homology to mediators of cholesterol homeostasis;Carstea;Science,1997

4. Cathepsin D protease mediates programmed cell death induced by interferon-γ, Fas/APO-1 and TNF-α;Deiss;Eur. Mol. Biol. Org.,1996

5. Pharmacological and genetic modifications of somatic cholesterol do not substantially alter the course of CNS disease in Niemann–Pick C mice;Erickson;J. Inherit. Metab. Dis.,2000

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